The mechanisms of neuroprotection are still largely incompletely known. We report here some findings on the role of autophagy, a cellular process by which unwanted/damaged organelles or proteins are eliminated, in the regulation of the cellular levels of BCL-2, one of the most widely investigated proteins that protects cells from death.
Autophagy regulates the post-translational cleavage of BCL-2 and promotes neuronal survival
LOSSI, Laura;GAMBINO, Graziana;SALIO, Chiara;MERIGHI, Adalberto
2010-01-01
Abstract
The mechanisms of neuroprotection are still largely incompletely known. We report here some findings on the role of autophagy, a cellular process by which unwanted/damaged organelles or proteins are eliminated, in the regulation of the cellular levels of BCL-2, one of the most widely investigated proteins that protects cells from death.
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